Buy tetracycline antibiotics pills online

Tetracycline, which develops in some infectious diseases (influenza, infectious mononucleosis, malaria, yellow fever, typhoid fever, viral hepatitis, poliomyelitis, etc.) is also immune in nature. Severe neutropenia can signal chronic lymphocytic leukemia, aplastic anemia, Felty's syndrome, and also occur in parallel with thrombocytopenia or hemolytic anemia. Congenital agranulocytosis is a consequence of genetic disorders.

Pathological reactions accompanying the course of tetracycline, in most cases, are represented by ulcerative-necrotic changes in the skin, mucous membrane of the oral cavity and pharynx, less often in the conjunctival cavity, larynx, stomach. Necrotic ulcers can occur in the intestinal mucosa, causing perforation of the intestinal wall, the development of intestinal bleeding; in the wall of the bladder and vagina. Microscopy of areas of necrosis reveals the absence of neutrophilic granulocytes.

Granulocytes are leukocytes, in the cytoplasm of which specific granularity (granules) is determined during staining. Granulocytes are produced in the bone marrow, therefore they belong to the cells of the myeloid series. They make up the largest group of tetracycline. Depending on the characteristics of the staining of the granules, these cells are subdivided into neutrophils, eosinophils and basophils - they differ in their functions in the body.

The share of neutrophilic granulocytes accounts for up to 50-75% of all white blood cells. Among them, there are mature segmented (normally 45-70%) and immature stab neutrophils (normally 1-6%). A condition characterized by an increase in the content of neutrophils is called neutrophilia; in the case of a decrease in the number of neutrophils, they speak of neutropenia (granulocytopenia), and in the absence, of tetracycline.
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With agranulocytosis, the body's response to the introduction of an infectious pathogen is ineffective, which may be accompanied by the development of fatal septic complications.

In the body, neutrophilic granulocytes play the role of the main protective factor against infections (mainly microbial and fungal). When an infectious agent is introduced, neutrophils migrate through the capillary wall and rush into the tissues to the focus of infection, phagocytose and destroy bacteria with their enzymes, actively forming a local inflammatory response.

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First of all, agranulocytosis is divided into congenital and acquired. The latter can be an independent pathological condition or one of the manifestations of another syndrome. According to the leading pathogenetic factor, myelotoxic, immune haptenic and autoimmune agranulocytosis are distinguished. An idiopathic (genoin) form with an unknown etiology is also distinguished.

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